Assuming you mean viral load referring to mucuses or blood of the infected. Given that the human influenza virus’ entry requires the viral surface proteins (hemagglutinin (HA)) to bind to acids present on respiratory epithelial cells along with cleavage of HA by host cell proteases (enzymes that breakdown proteins) to facilitate membrane fusion. These trypsin-like proteases are mainly expressed in airway tissues, restricting influenza viral tissue response to the respiratory tract. I would say it would be highly unlikely for influenza viral replication existing in an environment lacking this crucial interaction let alone a low-pH environment like the GI tract

Tbf you are supposed use the signal before turning wheel

Otherwise I’d have to install a gui

It’s not really like they are storing DNA sequences anyways. They use a genotyping array which just reads ~650k single nucleotide polymorphisms (SNPs).

An analogy would be 23andme has a 6.4mil page book of DNA for a single customer but they only know the position and letter of single character on every tenth page. Sure it’s enough to identify someone (You can confidently use 50 SNPs to identify these days) but it’s not like 23andme was ever storing a whole genome